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List of the antibodies used in Western blot analysis
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List of the antibodies used in Western blot analysis

Journal: American Journal of Physiology - Renal Physiology

Article Title: Genetic disruption of Npr1 depletes regulatory T cells and provokes high levels of proinflammatory cytokines and fibrosis in the kidneys of female mutant mice

doi: 10.1152/ajprenal.00621.2018

Figure Lengend Snippet: List of the antibodies used in Western blot analysis

Article Snippet: F : quantitative fluorescence intensity analysis of TLR4 protein expression was done by measuring intracellular fluorescence using MetaMorph software.

Techniques: Western Blot

Forward and reverse sequences of primers that were used to amplify specific mRNAs in the kidney tissue of natriuretic peptide receptor-A gene-targeted mice

Journal: American Journal of Physiology - Renal Physiology

Article Title: Genetic disruption of Npr1 depletes regulatory T cells and provokes high levels of proinflammatory cytokines and fibrosis in the kidneys of female mutant mice

doi: 10.1152/ajprenal.00621.2018

Figure Lengend Snippet: Forward and reverse sequences of primers that were used to amplify specific mRNAs in the kidney tissue of natriuretic peptide receptor-A gene-targeted mice

Article Snippet: F : quantitative fluorescence intensity analysis of TLR4 protein expression was done by measuring intracellular fluorescence using MetaMorph software.

Techniques: Sequencing

Analysis of Toll-like receptors (TLRs) and mammalian target of rapamycin (mTOR) expression in natriuretic peptide receptor-A (Npr1) gene-targeted kidneys in untreated mice and mice treated with rapamycin. A–C: relative mRNA expression of TLR2-, TLR4-, and TLR6-targeted genes normalized to β-actin mRNA in kidney tissues. D: renal expression levels of TLR4 protein were determined by Western blot analysis. E: immunofluorescence of TLR4 in rapamycin-treated and vehicle-treated Npr1 gene-targeted mouse kidneys. A marked increase in TLR4 expression occurred in Npr1 gene knockout kidneys but not in the kidneys of wild-type controls. Rapamycin decreased expression of TLR4 in tubular epithelial cells of kidneys. Red fluorescence indicates TLR4 protein expression; blue fluorescence indicates a nucleus stained with DAPI. F: quantitative fluorescence intensity analysis of TLR4 protein expression was done by measuring intracellular fluorescence using MetaMorph software. G: mTOR protein expression was determined by Western blot analysis. β-Actin was used as an internal control for normalization. Densitometric analyses of the respective bands were done with an Alpha Innotech phosphoimager. Values are expressed as means ± SE; n = 6/group. *P < 0.05, control and Npr1++/++ vs. rapamycin-treated Npr1++/++ mice; **P < 0.01, control Npr1+/+ and Npr1++/++ vs. rapamycin-treated Npr1+/+ and Npr1++/++ mice; ***P < 0.001, control Npr1+/− and Npr1−/− vs. rapamycin-treated Npr1+/−and Npr1−/− mice.

Journal: American Journal of Physiology - Renal Physiology

Article Title: Genetic disruption of Npr1 depletes regulatory T cells and provokes high levels of proinflammatory cytokines and fibrosis in the kidneys of female mutant mice

doi: 10.1152/ajprenal.00621.2018

Figure Lengend Snippet: Analysis of Toll-like receptors (TLRs) and mammalian target of rapamycin (mTOR) expression in natriuretic peptide receptor-A (Npr1) gene-targeted kidneys in untreated mice and mice treated with rapamycin. A–C: relative mRNA expression of TLR2-, TLR4-, and TLR6-targeted genes normalized to β-actin mRNA in kidney tissues. D: renal expression levels of TLR4 protein were determined by Western blot analysis. E: immunofluorescence of TLR4 in rapamycin-treated and vehicle-treated Npr1 gene-targeted mouse kidneys. A marked increase in TLR4 expression occurred in Npr1 gene knockout kidneys but not in the kidneys of wild-type controls. Rapamycin decreased expression of TLR4 in tubular epithelial cells of kidneys. Red fluorescence indicates TLR4 protein expression; blue fluorescence indicates a nucleus stained with DAPI. F: quantitative fluorescence intensity analysis of TLR4 protein expression was done by measuring intracellular fluorescence using MetaMorph software. G: mTOR protein expression was determined by Western blot analysis. β-Actin was used as an internal control for normalization. Densitometric analyses of the respective bands were done with an Alpha Innotech phosphoimager. Values are expressed as means ± SE; n = 6/group. *P < 0.05, control and Npr1++/++ vs. rapamycin-treated Npr1++/++ mice; **P < 0.01, control Npr1+/+ and Npr1++/++ vs. rapamycin-treated Npr1+/+ and Npr1++/++ mice; ***P < 0.001, control Npr1+/− and Npr1−/− vs. rapamycin-treated Npr1+/−and Npr1−/− mice.

Article Snippet: F : quantitative fluorescence intensity analysis of TLR4 protein expression was done by measuring intracellular fluorescence using MetaMorph software.

Techniques: Expressing, Western Blot, Immunofluorescence, Gene Knockout, Fluorescence, Staining, Software, Control